April 07, 2022

Thrombogenic Hypothesis of Heart Disease-Part 2

Articles by Dr. Erdman are for informational purposes, and are not to be taken as specific medical advice.

 

Continuing our exploration of the thrombogenic hypothesis for heart disease, we will look at what damages the endothelial lining besides what we talked about in the last article.

We will bring this discussion to current events.  Why is having an infection with the Wuhan virus or getting one of the jabs leading to heart issues?.

Anyone following the news now knows that getting those mRNA jabs have proven to result in a problem of post-jab heart issues. Recent data shows everyone under the age of 80 is at higher risk of dying from the jab than of Covid itself. For those under 18 years old, they are 51 times more likely to die from the jab than from the actual virus if unvaccinated!

But what is the mechanism that this virus and jab that causes heart issues?

When cells have a virus, they send out distress signals to the body saying to “come kill me, I’m diseased.” Well, the Covid virus enters the endothelial lining cells through the ACE2 receptor, and endothelial cells have lots of them. Once inside, it replicates and bursts out of the cell causing damage to arterial wall lining. There is your mechanism of action to induce blood clots!

Now, when tons of areas are experiencing this damage all at the same time, the body can’t keep up with repair and now you have strokes and heart attacks.

The Covid mRNA jabs are even worse! Now these cells are triggered to produce spike proteins, and these cells are sending out distress signals to be killed. Dr. Kendricks says, “You have to be very careful if you want to stick something into cells that then says to the immune system, ‘please, come and kill me,’ because That’s what the immune system will do.”

The other greatest cause of cell damage is smoking.  Smoke particles get out of your lungs, into your blood stream and they cause damage.

Luckily, as endothelial cells die off, the bone marrow replaces them by sending progenitor cells to cover the damaged area. Some smokers have enough repair going on when they are younger. But as you age, the repair mechanism slows down and it becomes more and more of a problem to repair damaged areas.

Repairing the glycocalyx is complicated. It has nitric oxide synthase which produces nitric oxide. Nitric oxide is a potent anti-clotting chemical. It also has albumin, a protein complex produced by the liver. Albumin contains the proteins necessary to repair the glycocalyx layer.

The glycocalyx layer can rapidly be destroyed, but it can also rapidly be repaired. Experiments have shown a completely stripped glycocalyx layer can be completely repaired in a single second! Supplements like glucosamine, chondroitin sulfate and MSM are helpful with this.

Nitro oxide (NO) is produced after vigorous exercise, and is found as nitrates in many foods like red beets, garlic, meat, dark chocolate, leafy greens, nuts and seeds, citrus fruits and red wine. NO makes blood platelets less sticky, therefore less likely to clot. Eat more of these products.

Avoid NSAIDS like ibuprofen, aspirin and naproxen. Get plenty of sunshine which triggers NO production. Avoid seed oils and processed foods. Lower your blood sugar and insulin levels. Address chronic stress and by all means quit smoking.

Cholesterol is not the cause of heart disease; arterial wall damage is!